Research
Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9)
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* Corresponding authors: Christoph Kleinschnitz christoph.kleinschnitz@mail.uni-wuerzburg.de - Sven G Meuth sven.meuth@gmx.de
- † Equal contributors
1 Westfaelische Wilhelms-University Muenster, Institute of Physiology I, Robert-Koch Str. 27a, 48149 Muenster, Germany
2 University of Wuerzburg, Department of Neurology, Josef-Schneider Str. 11, 97080 Wuerzburg, Germany
3 University of Muenster, Neurological Clinic - Inflammatory Disorders of the Central Nerveous System and Neurooncology, Mendelstr. 7, 48149 Muenster, Germany
Experimental & Translational Stroke Medicine 2010, 2:14 doi:10.1186/2040-7378-2-14
Published: 20 July 2010Additional files
Additional file 1:
Figure S1 - rCBF does not differ between wildtype mice and K2P9.1-/- mice. Determination of regional cerebral blood flow (rCBF) using Laser Doppler flowmetry before the occlusion of the middle cerebral artery (baseline), 10 min after the occlusion (ischemia) and again 10 min after the removal of the filament (reperfusion) in wildtype mice and K2P9.1-/- mice (n = 3/group). No significant differences in rCBF were observed between the two groups. One-way ANOVA, Bonferroni post hoc test.
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